"Methyl-1-Testosterone, does not aromatize, this means that Methyl-1-Testosterone does not convert to estrogen. Methyl-1-Testosterone is 5alpha-reduced androgen which prevents it from converting to estrogen, however due to the 5alpha-reduced it is a DHT-derivative. It is not testosterone, Methyl-1-Testerone is a 17aa version of
1-Testosterone, and
1-Testosterone is actually the 5alpha-reduced version of Boldenone, so by this we can
see that Methyl-1-Testerone is to
1-Testosterone, that Methandrostenolone is to Boldenone, and
1-Testosterone is to Testosterone as DHT is to Boldenone.
Some people have reported bloating and
other "estrogen like" effects; however these are actually progestin side effects. This leads us to the point that an ancillary SERM (Selective Estrogen Receptor Module) like Nolvadex would do no use because it is a competitor with estrogen with the estrogen receptors.
This leads me to another point! If for some obscure reason someone in questions is in fact running Methyl-1-Testosterone as a solitary androgen, they will shut down their HPTA which stops all natural Testosterone production. Testosterone plays many vital roles in the human body, including manages emotions and sex
drive. This will lead me to another point, the only way the male body can create estrogen is through the conversion or aromatization of testosterone, and with no testosterone in the body, and you will also leave yourself without estrogen. Estrogen even though it is termed as a female hormone it is essential to proper function of the male body. Now, for those of you who will recommend Nolvadex or a similar SERM for gynomastia, what will it do when estrogen and thus the estrogen receptors are not the cause of this gynomastia? Also as a side note, lets face it estrogen is essential to proper function, now everyone knows that SERMs have a high success rate of beating estrogen at the receptors, so if you have very little if any estrogen in your body, why would you want to block the small bit that you may still have?
Okay, okay, well your saying, if you're so smart what should we take? First of all I believe that your Methyl-1-Testerone should be stacked with an exogenous testosterone. However even if it is not, Methyl-1-Testosterone does have progestin like effects and it has been proven that where we find progestin we will find Prolactin, the two go hand and hand. So now we know that the cause of gynomastia or bloating or
other "estrogen like" side effects are actually Prolactin side effects, and thus we will need a Prolactin suppresser, and yes things that manage Prolactin actually suppress Prolactin, and I say this because aromatization inhibitors, do not suppress estrogen they
prevent the conversion to estrogen, and SERMs, do not suppress estrogen they compete with estrogen. So the weapon of choice to combat these side effects is actually Cabergoline."
so after reading this seems like i should be in the market for some Cabergoline...
Anyone interested in a 3 week M1T log?, startin it in 3 weeks..
..But ya nothing is really set in stone I might still stack it with 
